Thomas E. Cecere
· Assistant Professor of Biomedical Sciences and PathobiologyVirginia Tech · Department of Population Health Sciences
Active 2008–2026
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About
Thomas E. Cecere, DVM, PhD, DACVP–Anatomic Pathology, is an Associate Professor at the Virginia-Maryland College of Veterinary Medicine within Virginia Tech. His educational background includes a Bachelor of Science in Biochemistry from Virginia Tech, a Doctor of Veterinary Medicine from the same institution, a Master of Science in Veterinary Medicine with a focus on Anatomic Pathology from North Carolina State University, and a PhD in Viral Immunology from Virginia Tech. He is board-certified by the American College of Veterinary Pathologists in Anatomic Pathology. His professional experience encompasses roles as an Assistant Professor and now an Associate Professor of Anatomic Pathology at Virginia Tech, with prior training as a fellow in the NIH T32 Animal Model Research for Veterinarians Program and completing an Anatomic Pathology Residency at North Carolina State University. His research interests include swine infectious diseases, microbial immunology, diagnostic pathology, and pedagogy. Cecere has contributed to the field through various publications and is actively involved in professional memberships such as the American College of Veterinary Pathologists, the American Veterinary Medical Association, and the American Association of Veterinary Laboratory Diagnosticians.
Research topics
- Medicine
- Biology
- Internal medicine
- Immunology
- Pathology
- Chemistry
- Pharmacology
- Oncology
- Cancer research
- Urology
Selected publications
Journal of Alzheimer s Disease · 2020 · 103 citations
BACKGROUND: Porphyromonas gingivalis (P. gingivalis) and its gingipain virulence factors have been identified as pathogenic effectors in Alzheimer's disease (AD). In a recent study we demonstrated the presence of gingipains in over 90% of postmortem AD brains, with gingipains localizing to the cytoplasm of neurons. However, infection of neurons by P. gingivalis has not been previously reported. OBJECTIVE: To demonstrate intraneuronal P. gingivalis and gingipain expression in vitro after infectin…
Neuro-Oncology · 2020 · 42 citations
BACKGROUND: The interleukin-13 receptor alpha 2 (IL13RA2) and ephrin type A receptor 2 (EPHA2) are attractive therapeutic targets, being expressed in ~90% of canine and human gliomas, and absent in normal brain. Clinical trials using an earlier generation IL-13 based cytotoxin showed encouraging clinical effects in human glioma, but met with technical barriers associated with the convection-enhanced delivery (CED) method. In this study, IL-13 mutant and ephrin A1 (EFNA1)-based bacterial cytotoxi…
Severe SARS-CoV-2 Infection in a Cat with Hypertrophic Cardiomyopathy
Viruses · 2021 · 41 citations
) that presented with hypertrophic cardiomyopathy (HCM), a chronic heart condition that has been described as a comorbidity of COVID-19 in humans and that is prevalent in domestic cats. The lung and heart of the affected cat presented clear evidence of SARS-CoV-2 replication, with histological lesions similar to those observed in humans with COVID-19 with high infectious viral loads being recovered from these organs. The study highlights the potential impact of comorbidities on the outcome of SA…
Veterinary Surgery · 2021-05-10 · 22 citations
articleOpen accessOBJECTIVE: Design 3D printed skull contoured brain biopsy guides (3D-SCGs) from computed tomography (CT) or T1-weighted magnetic resonance imaging (T1W MRI). STUDY DESIGN: Feasibility study. SAMPLE POPULATION: Five beagle dog cadavers and two client-owned dogs with brain tumors. METHODS: Helical CT and T1W MRI were performed on cadavers. Planned target point was the head of the caudate nucleus. Three-dimensional-SCGs were created from CT and MRI using commercially available open-source software.…
Frontiers in Immunology · 2022-07-28 · 21 citations
articleOpen accessCommensal bacteria and the immune system have a close and strong relationship that maintains a balance to control inflammation. Alterations of the microbiota, known as dysbiosis, can direct reactivity to self-antigens not only in the intestinal mucosa but also at the systemic level. Our laboratory previously reported gut dysbiosis, particularly lower abundance of bacteria in the family Lactobacillaceae , in lupus-prone MRL/ lpr mice, a model of systemic autoimmunity. Restoring the microbiota wit…
Frequent coauthors
- 54 shared
Xin Luo
- 35 shared
Tanya LeRoith
Virginia Tech
- 34 shared
John H. Rossmeisl
Virginia Tech
- 32 shared
Christopher M. Reilly
- 30 shared
Qinghui Mu
Stanford University
- 29 shared
Leila Abdelhamid
Virginia Tech
- 28 shared
Xavier Cabana-Puig
- 22 shared
Michael Edwards
Virginia–Maryland College of Veterinary Medicine
Education
Other, Veterinary Medicine
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