Dave Bridges
· Associate Professor, Nutritional SciencesUniversity of Michigan · Nutritional Sciences
Active 2002–2026
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About
Professor Dave Bridges is an Associate Professor in the Department of Nutritional Sciences at the University of Michigan, a position he has held since September 1, 2022. The information provided does not include specific details about his research focus, background, or key contributions.
Research topics
- Internal medicine
- Medicine
- Biology
- Endocrinology
- Microbiology
- Chemistry
- Gastroenterology
- Biochemistry
- Genetics
- Immunology
Selected publications
Nature Communications · 2022 · 27 citations
Almost all effective treatments for non-alcoholic fatty liver disease (NAFLD) involve reduction of adiposity, which suggests the metabolic axis between liver and adipose tissue is essential to NAFLD development. Since excessive dietary sugar intake may be an initiating factor for NAFLD, we have characterized the metabolic effects of liquid sucrose intake at concentrations relevant to typical human consumption in mice. We report that sucrose intake induces sexually dimorphic effects in liver, adi…
Clinical and Translational Gastroenterology · 2021 · 20 citations
INTRODUCTION: Nonalcoholic fatty liver disease (NAFLD) has been increasingly reported among recipients of liver transplantation (LT). We aimed to identify clinical and genetic risk factors responsible for the development of early recurrent NAFLD in nonalcoholic steatohepatitis transplant recipients. METHODS: Forty-six total single nucleotide polymorphisms with known association with NAFLD were tested among both recipient and donor liver samples in 66 LT recipients with nonalcoholic steatohepatit…
AJP Endocrinology and Metabolism · 2020 · 18 citations
. We monitored maternal health and offspring growth during the lactation window, as well as adult glucose homeostasis through in vivo testing. At necropsy we assessed pancreas and adipocyte morphology using histological and immunofluorescent staining techniques. We found that metformin exposure programmed male and female offspring to be leaner with a higher proportion of small adipocytes in the gonadal white adipose tissue (GWAT). Male, but not female, offspring had an improvement in glucose tol…
AJP Lung Cellular and Molecular Physiology · 2021 · 16 citations
clearance and augments blood dissemination by reducing neutrophil killing and ROI due to impaired glucose transport.
GDF15 Knockout Does Not Substantially Impact Perinatal Body Weight or Neonatal Outcomes in Mice
Endocrinology · 2024-10-22 · 4 citations
articleOpen accessSenior authorCorrespondingGrowth differentiation factor-15 (GDF15) increases in circulation during pregnancy and has been implicated in food intake, weight loss, complications of pregnancy, and metabolic illness. We used a Gdf15 knockout mouse model (Gdf15-/-) to assess the role of GDF15 in body weight regulation and food intake during pregnancy. We found that Gdf15-/- dams consumed a similar amount of food and gained comparable weight during the course of pregnancy compared with Gdf15+/+ dams. Insulin sensitivity on ges…
Recent grants
Regulation of Lipid Storage by mTORC1
NIH · $1.7M · 2016–2022
Frequent coauthors
- 61 shared
Alan R. Saltiel
Salk Institute for Biological Studies
- 55 shared
Innocence Harvey
Pennington Biomedical Research Center
- 53 shared
JeAnna R. Redd
University of Michigan–Ann Arbor
- 35 shared
Erin J. Stephenson
- 30 shared
Quynh T. Tran
Cystic Fibrosis Foundation
- 25 shared
Irit Hochberg
Rambam Health Care Campus
- 23 shared
Greg B. G. Moorhead
University of Calgary
- 23 shared
Joan C. Han
Labs
Not provided
Education
- 2005
Ph.D., Biological Sciences
University of Calgary
- 2000
B.Sc., Biological Sciences
University of Calgary
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