Brittany Allen-Petersen

· Associate Professor

Purdue University · Biological Sciences

Active 2007–2025

h-index14

Citations1.4k

Papers7150 last 5y

Funding$176k

Academic metrics are sourced from OpenAlex and public funding records; values may differ from Google Scholar.

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About

Brittany L. Allen-Petersen, Ph.D., is an Assistant Professor in the Department of Biological Sciences at Purdue University and a Co-leader of the Indiana University Pancreatic Cancer Working Group. She completed her Ph.D. in Cell and Developmental Biology at the University of Colorado Anschutz Medical Campus and her B.S. in Molecular, Cellular, and Developmental Biology at the University of Michigan. Prior to her current position, she was a Postdoctoral Fellow at Oregon Health & Science University. Her professional biography highlights her academic background and leadership roles within cancer research, particularly pancreatic cancer, at Purdue University and Indiana University.

Research topics

Biology
Genetics
Medicine
Cancer research
Cell biology
Internal medicine
Biochemistry

Selected publications

PP2A and cancer epigenetics: a therapeutic opportunity waiting to happen
NAR Cancer · 2022 · 26 citations
Senior authorCorresponding
The epigenetic state of chromatin is altered by regulators which influence gene expression in response to environmental stimuli. While several post-translational modifications contribute to chromatin accessibility and transcriptional programs, our understanding of the role that specific phosphorylation sites play is limited. In cancer, kinases and phosphatases are commonly deregulated resulting in increased oncogenic signaling and loss of epigenetic regulation. Aberrant epigenetic states are kno…
DOI
The Prolyl Isomerase PIN1 Affects Fibroblast Differentiation States and Cross-talk in Pancreatic Cancer
Cancer Research · 2025-09-18 · 1 citations
articleOpen access
The prolyl isomerase PIN1 is overexpressed in cancer and contributes to cancer cell-intrinsic phenotypes, including proliferation and migration. However, PIN1 may also function in stromal cells within the tumor microenvironment. In this study, we showed that PIN1 is a critical regulator of pancreatic stellate cell (PSC) state at baseline and in response to the myofibroblast-activating factor TGFβ. Loss or inhibition of PIN1 altered the epigenetic and transcriptional responses of PSCs to TGFβ, pr…
Publisher OA PDF DOI
Supplementary Dataset 9 from The Prolyl Isomerase PIN1 Affects Fibroblast Differentiation States and Cross-talk in Pancreatic Cancer
2025-12-15
articleOpen access
NABA_MATRISOME overlap with DEGs (FPKMs)
Publisher DOI
Supplementary Dataset 14 from The Prolyl Isomerase PIN1 Affects Fibroblast Differentiation States and Cross-talk in Pancreatic Cancer
2025-12-15
articleOpen access
GSEA_shSCR vs shPIN1_C2all
Publisher DOI
Supplementary Dataset 10 from The Prolyl Isomerase PIN1 Affects Fibroblast Differentiation States and Cross-talk in Pancreatic Cancer
2025-12-15
articleOpen access
NABA_ECM overlap with DEGs (FPKMs)
Publisher DOI

Recent grants

MYC is a critical downstream effector in KRAS-driven pancreatic cancer
NIH · $176k · 2015–2018

Frequent coauthors

Rosalie C. Sears
Oregon Health & Science University
53 shared
Brett C. Sheppard
University of Portland
24 shared
Jennifer P. Morton
Cancer Research UK
21 shared
Dale J. Christensen
21 shared
Mary C. Thoma
Oregon Health & Science University
21 shared
Goutham Narla
University of Michigan–Ann Arbor
18 shared
Charles D. Lopez
University of Portland
16 shared
Colin J. Daniel
University of Portland
16 shared

Labs

Allen-Petersen LabPI
Not provided

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