Raymond Carney

About

Professor Raymond Carney has previously taught at Stanford University, the University of Texas, and Middlebury College. He is the author of more than 10 books, including titles such as Cassavetes on Cassavetes, The Films of Mike Leigh: Embracing the World, John Cassavetes: The Adventure of Insecurity, American Vision: The Films of Frank Capra, Speaking the Language of Desire: The Films of Carl Dreyer, The Films of John Cassavetes: Pragmatism, Modernism, and the Movies, and a monograph on Shadows in the BFI Film Classics series. He serves as the general editor of the Cambridge Film Classics, published by Cambridge University Press. Professor Carney curated the Beat Culture and the New America show for the Whitney Museum of American Art and is regarded as one of the world’s leading authorities on independent film and American literature, painting, and pragmatic philosophy, having edited or contributed to many volumes on these subjects.

Research topics

• Biology
• Medicine
• Bioinformatics
• Genetics
• Internal medicine
• Psychology
• Psychiatry
• Pathology
• Neuroscience
• Gerontology

Selected publications

• New insights into the genetic etiology of Alzheimer’s disease and related dementias

  Nature Genetics · 2022 · 2403 citations

  Characterization of the genetic landscape of Alzheimer's disease (AD) and related dementias (ADD) provides a unique opportunity for a better understanding of the associated pathophysiological processes. We performed a two-stage genome-wide association study totaling 111,326 clinically diagnosed/'proxy' AD cases and 677,663 controls. We found 75 risk loci, of which 42 were new at the time of analysis. Pathway enrichment analyses confirmed the involvement of amyloid/tau pathways and highlighted mi…

  DOI

• Assessment of the genetic variance of late-onset Alzheimer's disease

  Neurobiology of Aging · 2016-03-03 · 255 citations

  articleOpen access

  Alzheimer's disease (AD) is a complex genetic disorder with no effective treatments. More than 20 common markers have been identified, which are associated with AD. Recently, several rare variants have been identified in Amyloid Precursor Protein (APP), Triggering Receptor Expressed On Myeloid Cells 2 (TREM2) and Unc-5 Netrin Receptor C (UNC5C) that affect risk for AD. Despite the many successes, the genetic architecture of AD remains unsolved. We used Genome-wide Complex Trait Analysis to (1) e…

  PublisherDOI

• Transethnic genome‐wide scan identifies novel Alzheimer's disease loci

  Alzheimer s & Dementia · 2017-02-06 · 210 citations

  articleOpen access

  Abstract Introduction Genetic loci for Alzheimer's disease (AD) have been identified in whites of European ancestry, but the genetic architecture of AD among other populations is less understood. Methods We conducted a transethnic genome‐wide association study (GWAS) for late‐onset AD in Stage 1 sample including whites of European Ancestry, African‐Americans, Japanese, and Israeli‐Arabs assembled by the Alzheimer's Disease Genetics Consortium. Suggestive results from Stage 1 from novel loci were…

  PublisherOA PDFDOI

• <i>ABCA7</i> frameshift deletion associated with Alzheimer disease in African Americans

  Neurology Genetics · 2016-05-18 · 107 citations

  articleOpen access

  OBJECTIVE: To identify a causative variant(s) that may contribute to Alzheimer disease (AD) in African Americans (AA) in the ATP-binding cassette, subfamily A (ABC1), member 7 (ABCA7) gene, a known risk factor for late-onset AD. METHODS: Custom capture sequencing was performed on ∼150 kb encompassing ABCA7 in 40 AA cases and 37 AA controls carrying the AA risk allele (rs115550680). Association testing was performed for an ABCA7 deletion identified in large AA data sets (discovery n = 1,068; repl…

  PublisherOA PDFDOI

• Segregation of a rare <i>TTC3</i> variant in an extended family with late-onset Alzheimer disease

  Neurology Genetics · 2016-01-15 · 46 citations

  articleOpen access

  OBJECTIVE: The genetic risk architecture of Alzheimer disease (AD) is complex with single pathogenic mutations leading to early-onset AD, while both rare and common genetic susceptibility variants contribute to the more widespread late-onset AD (LOAD); we sought to discover novel genes contributing to LOAD risk. METHODS: Whole-exome sequencing and genome-wide genotyping were performed on 11 affected individuals in an extended family with an apparent autosomal dominant pattern of LOAD. Variants o…

  PublisherOA PDFDOI

Frequent coauthors

• Margaret A. Pericak‐Vance

  Dr. John T. Macdonald Foundation

  307 shared

• Badri N. Vardarajan

  Columbia University Irving Medical Center

  278 shared

• Brian W. Kunkle

  University of Miami

  274 shared

• John R. Gilbert

  266 shared

• Eden R. Martin

  University of Miami

  262 shared

• Gary W. Beecham

  University of Miami

  258 shared

• Jonathan L. Haines

  Case Western Reserve University

  248 shared

• Richard Mayeux

  Columbia University

  240 shared

Education

• Ph.D.

  Stanford University

• M.S.

  University of Texas

• B.A.

  Middlebury College

Similar researchers at Boston University

• Christopher Andersonh-42