About

Joemy Ramsay is a faculty member at the Spencer Fox Eccles School of Medicine, primarily in the Department of Surgery, Division of Urology. She holds an MS and PhD, with her education including a BS from the University of Utah, an MS from the University of Washington, and a PhD from the University of Utah. Her research focuses on various aspects of reproductive health, fertility, and environmental exposures, including the impact of air pollution on cardiovascular and respiratory health, familial cancer risk, and male reproductive health. She has contributed to studies examining racial and socioeconomic disparities in trauma care, the effects of industrial air pollution on fertility, and the familial patterns of cancer risk in subfertile men. Her work also explores the influence of environmental and genetic factors on fertility outcomes, perinatal health, and the health of cancer survivors. Dr. Ramsay's research is characterized by a strong emphasis on population-based studies, utilizing large datasets such as the Utah Population Database, and she has authored numerous publications in reputable journals related to urology, epidemiology, and environmental health.

Research topics

• Pediatrics
• Medicine
• Internal medicine
• Demography
• Family medicine
• Immunology
• Gerontology

Selected publications

• IP81-02 GENITOURINARY MEDICINE: EARLY RESULTS OF AN EMERGING PRIMARY CARE SUBSPECIALTY

  The Journal of Urology · 2026-04-27

  article
  PublisherDOI

• Impact of HCV infection on male reproductive hormones; a systematic review and meta-analysis

  International Journal of Impotence Research · 2026-05-20

  article
  PublisherDOI

• Epigenetic Signatures in Family Clusters: Links Between Male Fertility and Autism Risk

  Urology · 2026-04-01

  article
  PublisherDOI

• PD11-12 BOWEL-RELATED QUALITY OF LIFE OUTCOMES FOLLOWING BOWEL-CONTAINING URINARY RECONSTRUCTION IN PATIENTS WITH MIXED ETIOLOGIES OF BLADDER DYSFUNCTION

  The Journal of Urology · 2026-04-27

  article
  PublisherDOI

• IP09-26 RISK OF HYPOSPADIAS INCREASES WITH PRECONCEPTION AND PRENATAL EXPOSURES TO INDUSTRIAL CHEMICAL AIR POLLUTION

  The Journal of Urology · 2025-04-08

  article1st authorCorresponding
  PublisherDOI

• Health Insurance Coverage Among Adolescent and Young Adult Cancer Survivors Receiving Health Care before and During the COVID-19 Pandemic

  Journal of Adolescent and Young Adult Oncology · 2025-02-10

  articleOpen access

  We investigated insurance coverage among adolescents and young adults (AYA) with cancer before and during the COVID-19 pandemic. AYAs diagnosed with cancer 15–39 years of age were identified using Utah Cancer Registry records and linked with University of Utah electronic health records. Poisson models calculated incidence rate ratios (IRRs) of health insurance coverage during pre-pandemic (11/4/2017–3/5/2020; n = 2,140) and pandemic (3/6/2020–7/6/2022; n = 1,894) periods. Prior to the pandemic, insurance gaps were higher (pre-pandemic = 16.40%, pandemic = 13.73%; IRR = 0.84, 95%CI = 0.71–0.98); more AYAs had continuous public insurance during the pandemic (pre-pandemic = 8.60%, pandemic = 10.98%; IRR = 1.28, 95%CI = 1.05–1.56). Research is needed on the durability of pandemic relief programs on insurance coverage among AYA cancer survivors.

  PublisherOA PDFDOI

• DESCRIBING PATTERNS OF INCREASED RISK FOR AUTISM SPECTRUM DISORDER (ASD) IN FAMILIES OF SUBFERTILE MEN USING POPULATION PEDIGREE DATA

  Fertility and Sterility · 2025-12-01

  articleOpen access1st authorCorresponding
  PublisherOA PDFDOI

• Environmental PM2.5 Exposure and Socioeconomic Deprivation Are Independent Risk Factors for Impaired Semen Parameters

  Urology · 2025-08-01 · 1 citations

  article
  PublisherDOI

• Centralized Health and Exposomic Resource (C-HER): Analytic and AI-Ready Data for External Exposomic Research

  ArXiv.org · 2025-11-04

  preprintOpen access

  The Centralized Health and Exposomic Resource (C-HER) project has identified, profiled, spatially indexed, and stored over 30 external exposomic datasets. The resulting analytic and AI-ready data (AAIRD) provides a significant opportunity to develop an integrated picture of the exposome for health research. The exposome is a conceptual framework designed to guide the study of the complex environmental and genetic factors that together shape human health. Few composite measures of the exposome exist due to the high dimensionality of exposure data, multimodal data sources, and varying spatiotemporal scales. We develop a data engineering solution that overcomes the challenges of spatio-temporal linkage in this field. We provide examples of how environmental data can be combined to characterize a region, model air pollution, or provide indicators for cancer research. The development of AAIRD will allow future studies to use ML and deep learning methods to generate spatial and contextual exposure data for disease prediction.

  PublisherOA PDFDOI

• A scoping review of the role of heritability and environmental exposures in the development and severity of benign prostatic hyperplasia

  Translational Andrology and Urology · 2025-08-01 · 1 citations

  reviewOpen access

  Background: Benign prostatic hyperplasia (BPH) is a common condition among aging men, significantly affecting quality of life and contributing to a substantial healthcare burden. The pathogenesis of BPH is strongly influenced by genetic factors, with heritability estimates showing a wide range from 20% to 83%. Emerging evidence also highlights the critical role of environmental exposures, including endocrine-disrupting chemicals (EDCs), in BPH risk, progression, and therapeutic response. This review synthesizes current knowledge on genetic and environmental determinants of BPH pathogenesis, severity, and management. Methods: A scoping review of the literature was conducted using the databases PubMed, Scopus, and Web of Science. Relevant studies on genetic predisposition, environmental exposures, and their contributions to BPH were analyzed. Data from epidemiological studies, genome-wide association studies (GWAS), familial aggregation analyses, and research on environmental exposures were integrated to provide an understanding of these factors and BPH pathogenesis. Results: , associated with prostate volume and aggressive BPH phenotypes. Polygenic risk scores offer promising applications in identifying individuals at high risk for severe BPH. Environmental exposures, particularly to EDCs such as bisphenol A (BPA), bisphenol S (BPS), and bisphenol AF (BPAF), were found to disrupt hormonal regulation, contributing to prostatic hyperplasia. Air pollution, primarily particulate matter, exacerbates prostate inflammation and hyperplasia, with regional differences in BPH symptom severity correlating with air quality. Lifestyle factors, including high-fat diets and sedentary behaviors, further modulate disease severity. Conclusions: The development and progression of BPH are shaped by a complex interplay of genetic and environmental factors. EDCs contribute significantly to prostatic hyperplasia, while heritable factors influence disease onset, severity, and response to treatment. Integrating genetic risk profiling and environmental exposure assessments into clinical practice holds the potential to enhance BPH management and personalized therapeutic strategies.

  PublisherDOI

Frequent coauthors

• Anne C. Kirchhoff

  59 shared

• Judy Y. Ou

  University of Utah

  55 shared

• Heydon K. Kaddas

  44 shared

• Heidi A. Hanson

  University of Utah

  40 shared

• Douglas Fair

  University of Utah

  33 shared

• Echo L. Warner

  University of Utah

  30 shared

• Deanna Kepka

  29 shared

• Joshua J. Horns

  University of Utah

  22 shared

Labs

• Joemy Ramsay LabPI

Education

• Ph.D.

  University of Utah

• M.S.

  University of Utah