About

Dr. Farah Daccueil is a Nephrology specialist and a Clinical Associate Professor in Medicine at Stony Brook University. She completed her fellowship in Nephrology at NYU Langone Health - Winthrop University Hospital in 2017 and her residency in General Medicine at the same institution in 2015. She earned her medical degree from SUNY Syracuse Medical School in 2012. Dr. Daccueil is board certified in Nephrology and Internal Medicine by the American Board of Internal Medicine. She speaks multiple languages including Spanish, Creole - Haitian, French, and English. Her professional focus is on nephrology, and she is actively involved in clinical practice and teaching at Stony Brook Medicine.

Research topics

• Medicine
• Internal medicine
• Gastroenterology
• Oncology
• Urology
• Immunology
• Intensive care medicine
• Medical emergency
• Pathology
• Chemistry
• Pediatrics
• Endocrinology

Selected publications

• Personalized Approach to Antineutrophil Cytoplasmic Antibody-Associated Vasculitis Leading to Rapidly Progressive Glomerulonephritis Treatment with Glucocorticoids, Plasma Exchange, and Hemodialysis

  Journal of the American Society of Nephrology · 2025-10-01

  articleSenior author
  PublisherDOI

• Glomerular Chaos: Diffuse Proliferative Glomerulonephritis in CKD

  Journal of the American Society of Nephrology · 2025-10-01

  articleSenior author

  Introduction: Diffuse proliferative glomerulonephritis (DPGN) is a kidney disorder characterized by the activation of the alternative complement pathway, often associated with infection-related and C3 deposits. The clinical outcomes and onset of DPGN are variable and may include complete recovery, progression to chronic kidney disease, or advancement to end-stage renal disease. Case Description: A 74-year-old man with diabetes, hypertension, and stage 3 chronic kidney disease (baseline creatinine 1.8 mg/dL) presented with weakness. Labs showed mild renal impairment (creatinine 2.0 mg/dL), elevated CPK, BNP, WBC, and normal lipid profile, HbA1c, and lactate. Blood cultures grew methicillin-sensitive Staphylococcus aureus, and he was treated with IV oxacillin. Initially stable, his creatinine rose to 7.6 mg/dL with oliguria and uremic symptoms, requiring dialysis. Urinalysis showed proteinuria, hematuria, and glucosuria. Oxacillin-induced acute interstitial nephritis was suspected, and steroids were started. However, serologies were negative, and kidney biopsy revealed C3-dominant, infection-associated diffuse proliferative glomerulonephritis with acute tubular necrosis, not interstitial nephritis. Discussion: DPGN with infection-associated C3 deposits is characterized by diffuse involvement of most glomeruli, proliferation of mesangial and endothelial cells, and prominent deposition of C3 complement within the glomeruli. This condition is most commonly linked to Group A Streptococcus, Staphylococcus aureus infections, Escherichia coli, and Pseudomonas species; viral and fungal causes are less common. DPGN typically affects children, young adults, and the elderly, with a male predominance and a higher incidence in underserved populations. Prognosis is generally worse in individuals with underlying comorbidities like diabetes mellitus, autoimmune diseases, or pre-existing chronic kidney disease. Histological findings such as crescent formation on kidney biopsy and a lower estimated glomerular filtration rate at diagnosis are also associated with poorer outcomes. Prompt and effective treatment of the underlying infection is crucial for preventing progression of the glomerulonephritis. Early diagnosis and targeted therapy help resolve the primary infectious cause and minimize renal damage, improving long-term outcomes and preserving kidney function.

  PublisherDOI

• Obstructive Nephropathy Unmasking a Suicide Attempt with Ethylene Glycol Poisoning

  Journal of the American Society of Nephrology · 2024

  • Medicine
  • Medical emergency
  • Intensive care medicine

  Introduction: Ethylene glycol (EG) poisoning is a life-threatening condition that requires early diagnosis to prevent complications and death. This is a patient with obstructive nephropathy from EG poisoning who successfully recovered after fomepizole and renal replacement therapy. Case Description: 67-year-old male presented with acute encephalopathy. Vitals showed blood pressure 180/74 mmHg, respirations 22, and temperature 35.5 °C. Laboratory included K 5.8 mg/dL, bicarbonate <6 mmol/L, serum creatinine 2.2 mg/dL, BUN 22 mg/dL, lactic acid 12.7 mmol/L, urine toxicology, and ethanol normal. CT head non-revealing and CT abdomen and pelvis bilateral obstructing calculus resulting in hydronephrosis. He was taken for urgent bilateral stent placement by urology and admitted to MICU for urosepsis. Nephrology was consulted for persistent acidosis, acute kidney injury, and electrolyte derangements. Nephrology investigation showed anion gap 35 mmol/L, osmolar gap 100, ethylene glycol level 293 mg/dL, pH 7.1. Formal ethylene glycol toxicity was made, and oxalate monohydrate stones found; He was treated with IV fluids, vasopressors, IV antibiotics, fomepizole, and continuous renal replacement therapy. He fully recovered and was discharged to inpatient psychiatry after confessing suicidal attempt. Discussion: EG is a dangerous sweet-tasting, colorless, and odorless liquid used as an industrial compound. It causes significant morbidity and mortality if left untreated. More than 5,000 cases of intentional or unintentional toxicity are reported in the United States yearly, affecting mostly adult men. EG is broken down into glycolic acid, oxalic acid, glyoxylic acid, and glycolaldehyde causing kidney injury, central nervous system, and cardiopulmonary compromise. The glycolic acid is known to cause severe high anion gap metabolic acidosis, high osmolar gap, and oxalate precipitation causing kidney injury. In our case, he had acute encephalopathy, severe acidosis, high osmolar gap, and obstructive nephropathy with bilateral oxalate monohydrate stones unmasking an unknown suicide attempt. EG poisoning is challenging to diagnose and manage due to non-specific symptoms, limitations on direct EG quantification methods, and the shortened duration of an elevated osmolar gap in chronic alcohol abusers. High suspicion can prevent irreversible, and life-threatening complications.

  PublisherDOI

• Don't Get Burned Twice by the Same Post-transplant Lymphoproliferative Disorder (PTLD): A Case Highlighting the Progression and Advancement in PTLD Management in One Transplant Patient

  Journal of the American Society of Nephrology · 2024

  • Medicine
  • Pediatrics
  • Internal medicine
  PublisherDOI

• Beyond Anemia of CKD: A Tale of Discovering Post-transplant Lymphoproliferative Disorder by Colonoscopy

  Journal of the American Society of Nephrology · 2024-10-01

  article

  Introduction: In young patients seldom are drops in hemoglobin investigated with colonoscopy. In chronic kidney disease (CKD) patients, anemia is attributed to CKD and further testing is often met with resistance especially in younger population. Our rare case describes how a nephrologists referral for anemia diagnoses post-transplant lymphoproliferative disorder (PTLD) in the form of colon lymphoma. This highlights the important role nephrologist play in caring for transplant patients. Case Description: A 31-year-old male with congenital nephrotic syndrome (Finnish type) post living related kidney transplant 3/30/2006 is referred by nephrologist for inpatient workup of diarrhea and anemia. Exam revealed pale and toxic appearing young male with admission labs of Na 125, K 5.9, Cl 96, HCO3 16, BUN 73, Cr 3.64 and WBC 12.99, Hb 6.6, Hct 20.8 and PLT 403. Stool pathogen was positive for Yersinia Enterocolitica and E.coli (EPEC). CT imaging revealed pleural effusion and no lymphadenopathy. Although patient responded to transfusion, nephrology team had extensive discussion to pursue endoscopy which gastroenterology finally agreed. EGD/Colonoscopy revealed multiple polypoid non-obstructing medium sized masses in descending colon, transverse colon and ascending colon and multiple superficially ulcerated nodules in recto-sigmoid and sigmoid colon. Pathology diagnosed diffuse large B-cell lymphoma (DLBCL) negative for CD-20. Immunosuppression was reduced and patient was discharged however outpatient PET found mesenteric lymphadenopathy. Even though oncology initiated chemotherapy, the patient suffered septic shock due to enterotoxigenic E.coli after first treatment and expired. Discussion: Immunosuppressed kidney transplant recipients are at higher risk of developing malignancy including gastroenterology cancer. However current guidelines for testing and screening do not differ between the transplant and general population. Therefore continued sense of awareness and advocacy for screening is important. Evidence based guidelines in this population is needed to improve life expectancy in kidney transplant patients.

  PublisherDOI

• Unique Case of Hemosiderosis-Induced Ascites in Kidney Transplant Recipient

  Journal of the American Society of Nephrology · 2024

  Senior authorCorresponding
  • Medicine
  • Urology
  • Internal medicine

  Introduction: Iron overload or hemosiderosis is often a frequent complication from intermittent blood transfusions. Anemia of end stage kidney disease (ESKD) is managed with IV iron and erythropoietin stimulating agents (ESAs) and these patients are at risk of iron overload, especially those with increased dialysis vintage. Here we describe a unique case of recurrent ascites caused by secondary hemosiderosis in a kidney transplant recipient who was on Peritoneal Dialysis (PD) for 12 years. Case Description: A 61-year-old female with ESKD secondary to presumed Hypertension underwent 2nd deceased donor renal transplant(DDRT) in 2023. She was on PD from 2011 to 2023. She required 8 units of PRBC’s peri-operatively. Nadir creatinine 0.77 mg/dl. Post-transplant course complicated with COVID PNA, transplant renal artery stenosis requiring stent placement. She developed ascites three months post-transplant requiring frequent large volume paracentesis which was contributed to hepatic congestion in setting of heart failure with preserved ejection fraction (HFpEF). Seven months post-transplant she developed AKI in setting of E.coli bacteremia, CMV viremia and a transplant renal biopsy was performed showing focal endothelialitis, suggesting T cell mediated rejection, CMV nephritis and renal hemosiderosis and about 40% IFTA. Labs also showed iron levels of 15, Ferritin of 1400, TSAT 27%, AST/ALT normal, with elevated ALP 130. MRI liver was done which showed mild diffuse gain of signal in liver and spleen. A liver Biopsy was also performed which showed nodular regenerative hyperplasia and 1-2+ iron staining in Kupffer’s cells + hepatocytes and no evidence of fibrosis. Given liver biopsy findings and multiple transfusion history, it can be hypothesized that recurrent ascites was likely to secondary hemosiderosis rather than HFpEF. Patient’s AKI recovered with treatment of underlying infection. Of recent, she has not required paracentesis. Discussion: Here we describe an interesting case of a transplant patient with high dialysis vintage who developed recurrent ascites due to renal hemosiderosis. It is possible that long term IV iron exposure and multiple transfusions immediately post-transplant lead to secondary hemosiderosis. Long term PD could be contributing to development of ascites as well.

  PublisherDOI

• Is It Rhabdomyolysis or Is It Lupus? A Case of Dual Presentation of New-Onset Lupus

  Journal of the American Society of Nephrology · 2023-11-01

  articleSenior author

  Journal of the American Society of Nephrology 34(11S):p 276, November 2023. | DOI: 10.1681/ASN.20233411S1276a

  PublisherDOI

• Supplementary Material for: Association of Proteinuria and Hematuria with Acute Kidney Injury and Mortality in Hospitalized Patients with COVID-19

  Figshare · 2020-01-01

  datasetOpen access

  <b><i>Introduction:</i></b> Acute kidney injury (AKI) is strongly associated with poor outcomes in hospitalized patients with coronavirus disease 2019 (COVID-19), but data on the association of proteinuria and hematuria are limited to non-US populations. In addition, admission and in-hospital measures for kidney abnormalities have not been studied separately. <b><i>Methods:</i></b> This retrospective cohort study aimed to analyze these associations in 321 patients sequentially admitted between March 7, 2020 and April 1, 2020 at Stony Brook University Medical Center, New York. We investigated the association of proteinuria, hematuria, and AKI with outcomes of inflammation, intensive care unit (ICU) admission, invasive mechanical ventilation (IMV), and in-hospital death. We used ANOVA, <i>t</i> test, χ² test, and Fisher’s exact test for bivariate analyses and logistic regression for multivariable analysis. <b><i>Results:</i></b> Three hundred patients met the inclusion criteria for the study cohort. Multivariable analysis demonstrated that admission proteinuria was significantly associated with risk of in-hospital AKI (OR 4.71, 95% CI 1.28–17.38), while admission hematuria was associated with ICU admission (OR 4.56, 95% CI 1.12–18.64), IMV (OR 8.79, 95% CI 2.08–37.00), and death (OR 18.03, 95% CI 2.84–114.57). During hospitalization, de novo proteinuria was significantly associated with increased risk of death (OR 8.94, 95% CI 1.19–114.4, <i>p</i> = 0.04). In-hospital AKI increased (OR 27.14, 95% CI 4.44–240.17) while recovery from in-hospital AKI decreased the risk of death (OR 0.001, 95% CI 0.001–0.06). <b><i>Conclusion:</i></b> Proteinuria and hematuria both at the time of admission and during hospitalization are associated with adverse clinical outcomes in hospitalized patients with COVID-19.

  PublisherDOI

• Association of Proteinuria and Hematuria with Acute Kidney Injury and Mortality in Hospitalized Patients with COVID-19

  Kidney & Blood Pressure Research · 2020 · 60 citations

  • Medicine
  • Internal medicine
  • Gastroenterology

  INTRODUCTION: Acute kidney injury (AKI) is strongly associated with poor outcomes in hospitalized patients with coronavirus disease 2019 (COVID-19), but data on the association of proteinuria and hematuria are limited to non-US populations. In addition, admission and in-hospital measures for kidney abnormalities have not been studied separately. METHODS: This retrospective cohort study aimed to analyze these associations in 321 patients sequentially admitted between March 7, 2020 and April 1, 2020 at Stony Brook University Medical Center, New York. We investigated the association of proteinuria, hematuria, and AKI with outcomes of inflammation, intensive care unit (ICU) admission, invasive mechanical ventilation (IMV), and in-hospital death. We used ANOVA, t test, χ2 test, and Fisher's exact test for bivariate analyses and logistic regression for multivariable analysis. RESULTS: Three hundred patients met the inclusion criteria for the study cohort. Multivariable analysis demonstrated that admission proteinuria was significantly associated with risk of in-hospital AKI (OR 4.71, 95% CI 1.28-17.38), while admission hematuria was associated with ICU admission (OR 4.56, 95% CI 1.12-18.64), IMV (OR 8.79, 95% CI 2.08-37.00), and death (OR 18.03, 95% CI 2.84-114.57). During hospitalization, de novo proteinuria was significantly associated with increased risk of death (OR 8.94, 95% CI 1.19-114.4, p = 0.04). In-hospital AKI increased (OR 27.14, 95% CI 4.44-240.17) while recovery from in-hospital AKI decreased the risk of death (OR 0.001, 95% CI 0.001-0.06). CONCLUSION: Proteinuria and hematuria both at the time of admission and during hospitalization are associated with adverse clinical outcomes in hospitalized patients with COVID-19.

  PublisherDOI

• Optimizing On-the-Go Learning Utilizing Short Modules on Topics Related to Nephrology

  Journal of the American Society of Nephrology · 2020-10-01

  articleSenior author

  Background: Residents and medical students are expected to formulate evidencebased treatment plans by keeping up with the most recent guidelines but that can be difficult given our schedules. In such circumstances, it is ideal to learn bite-sized pieces on the go. Methods: We created two modules about Phosphorus Binders and Oral Hypoglycemic Agents using a friendly graphic interface called Prezi. These modules were estimated to take 10-15 minutes and were accompanied by a total of seven content related questions that were compiled from Uworld Step 3 Question Bank, John Hopkins Primary Modules, and hospital courses of patients seen at Stony Brook. Survey monkey was utilized to create the pre and posttest. The modules were sent to third and fourth year medical students. Data was collected for 10 days. Results: Wilcoxon signed rank test was utilized to evaluate the effectiveness of the modules. Unfortunately, only six students completed the hypoglycemic module of which two had no improvement in scores, so no statistical significance was achieved. However, four of the six students had improvement in posttest scores by at least one point. Improvement in the posttest scores for the phosphorus module was significant as of the nine students who completed the phosphorus module, eight had an improvement by at least 1 point (W 36, p=0.008). Conclusions: The observation of improved posttest scores for the phosphorus module supports the use of short lessons using a friendly graphic interface such as Prezi.

  PublisherDOI

Frequent coauthors

• Nobuyuki Miyawaki

  New York University

  26 shared

• Macaulay Onuigbo

  25 shared

• Lia Perez

  Moffitt Cancer Center

  25 shared

• M. Joseph

  25 shared

• Kenneth R. Wilund

  University of Arizona

  25 shared

• Tobias Pflederer

  25 shared

• Hao F

  25 shared

• Abdelhafiz I. Bashir

  University of Ha'il

  20 shared

Education

• M.D.

  SUNY @ Syracuse Medical School

  2012

• M.D., IM-General Medicine

  NYU Langone Health - Winthrop University Hospital

  2015

• M.D., IM-Nephrology

  NYU Langone Health - Winthrop University Hospital

  2017