Roger E. Turbin
· ProfessorVerifiedRutgers University · Ophthalmology and Visual Science
Active 1998–2025
About
Dr. Roger E. Turbin is a highly distinguished Professor of Ophthalmology at Rutgers New Jersey Medical School. He completed his undergraduate degree from Washington University in St. Louis, MO, where he also obtained his Doctor of Medicine in 1993. Dr. Turbin completed an internship at the University of Utah School of Medicine and his residency at NYU. He is a highly respected expert and leader in the field of neuro-ophthalmology as well as orbital and oculoplastic surgery. Dr. Turbin often serves as an attending on-call trauma surgeon for plastic and neuro-ophthalmology at University Hospital in Newark, NJ. His medical licensure includes certifications from the American Board of Ophthalmology in Neuro-ophthalmology and Ophthalmic Plastic/Reconstructive & Orbital Surgery. His research and clinical expertise encompass a range of topics including meningioma, pseudotumor cerebri, temporal arteritis, thyroid disease, and emerging techniques in orbital surgery.
Research topics
- Surgery
- Medicine
- Ophthalmology
Selected publications
A Challenging Case of Progressive Bilateral Optic Neuropathy
Journal of Neuro-Ophthalmology · 2025-10-23
articleAnnals of Diagnostic Pathology · 2024-09-27 · 1 citations
articleChild s Nervous System · 2024-04-22
reviewIatrogenic Pseudo-Duane Retraction Syndrome Following Orbitozygomatic Craniotomy
Journal of Pediatric Ophthalmology & Strabismus · 2024-01-01
articleSenior authorRestrictive strabismus is a known complication of orbitozygomatic craniotomy. However, a pseudo-Duane syndrome has not been described following this procedure. The authors describe a 58-year-old woman who after craniotomy developed incomitant left exotropia with an adduction deficit; the globe retracted and palpebral fissure narrowed with attempted ocular adduction. [ J Pediatr Ophthalmol Strabismus . 2024;61(1):e7–e10.]
Can You Tell Me About Stereotactic Image Guidance and How It Is Used in Orbital Surgery?
2024-05-30
book-chapter1st authorCorrespondingOphthalmology · 2024-10-22 · 1 citations
articleOpen accessHow Do I Manage an Orbital Apex Syndrome?
2024-05-28
book-chapter1st authorCorrespondingA 35-year-old man with diabetes mellitus and a history of diabetic ketoacidosis (DKA) presents with new onset painful ophthalmoplegia and loss of vision in his left eye. There might be a bit of proptosis as well. What should I be worried about and how soon does he need to be seen?
What are the Signs and Symptoms of an Orbital Fracture?
2024-05-28
book-chapter1st authorCorrespondingChildren with orbital fractures may present with signs and symptoms similar to those in adults. However, compared with their adult counterparts, children more commonly have clinically significant motility problems, despite more limited signs of bruising (white-eyed blowout 1 ), and typically require earlier intervention to avoid permanent ocular misalignment. This discussion will be limited to the orbital floor fracture in the setting of limited facial trauma, rather than to more extensive closed head trauma or penetrating orbitocranial injury. More severe injuries predispose to complex fractures, including nasoethmoidal complex fractures, zygomatico-maxillary tripod fractures, skull base fractures involving the orbital canal, and Le Forte midfacial fractures. Penetrating injury also represents a topic with which clinicians ought to be familiar, given the difficulty and occasionally failure of computed tomography (CT) imaging to recognize orbitocranial penetration as well as occult foreign bodies composed of dry and hydrated wood. The latter two may have CT signal indistinguishable from air or surrounding tissue, respectively.
Rapid response of thyroid eye disease, peripheral edema, and acropathy to teprotumumab infusion
American Journal of Ophthalmology Case Reports · 2024-03-02 · 4 citations
articleOpen accessSenior authorCorrespondingPurpose: We present a case of rapid improvement in symptoms of thyroid eye disease and amelioration of worsening peripheral edema and acropathy with infusion of teprotumumab, a monoclonal antibody targeting the insulin-like growth factor-1 receptor. Observations: A 66 year old female with history of Hashimoto thyroiditis developed progressive thyroid eye disease (TED), peripheral edema, and acropathy attributable to acute Graves disease. Her signs and symptoms, refractory to oral steroid and diuretic therapy, rapidly improved following a standard dosing regimen of teprotumumab (one infusion 10 mg/kg then seven infusions 20 mg/kg) to resolution. Conclusions & importance: Teprotumumab, a monoclonal antibody targeting the insulin-like growth factor-1 receptor, is the first medication approved by the FDA for use in TED. Teprotumumab may contribute to the treatment of extraocular manifestations of Graves disease, chief among these peripheral soft tissue manifestations.
What Is the Best Surgical Intervention for Patients With Idiopathic Intracranial Hypertension?
Journal of Neuro-Ophthalmology · 2023-04-20 · 3 citations
articleSubramanian, Prem S. MD, PhD; Turbin, Roger E. MD, FACS; Dinkin, Marc J. MD; Lee, Andrew G. MD; Van Stavern, Gregory P. MDEditor(s): Lee, Andrew G. MD; Van Stavern, Gregory P. MD Author Information
Frequent coauthors
- 115 shared
Larry Frohman
Rutgers New Jersey Medical School
- 100 shared
Paul D. Langer
Rutgers, The State University of New Jersey
- 82 shared
Luis M. Tumialán
Barrow Neurological Institute
- 81 shared
Zachary Litvack
Neuroscience Institute
- 81 shared
Nelson M. Oyesiku
- 81 shared
Chirag G. Patil
Cedars-Sinai Medical Center
- 81 shared
Manish K. Aghi
- 81 shared
Gabriel Zada
Education
- 1989
B.A.
Washington University
- 1989
B.S.
Washington University
- 1993
M.D.
University of Washington School of Medicine
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